Kathleen Folbigg: Pardoning the mother of a baby who died is "a victory for science."

Kathleen Folbigg

Australian woman who was sentenced to death for murdering her four infants says the decision to release her after 20 years is "a victory for science" and "truth.".

After an investigation upheld fresh evidence that gave her convictions "reasonable doubt," Kathleen Folbigg was allowed to leave prison on Monday.

Evidence later showed that she was not guilty of suffocating her kids, but rather that they suffered from rare genetic abnormalities.

The 55-year-old said being free had left her "humbled" and "grateful.".

She stated in a video statement that she has been grieving for her children for the past 20 years that she has been incarcerated.

A campaign to have her case reviewed was recently waged, and Ms. Folbigg thanked her friends and supporters for their efforts.

Without them, she "would not have survived this entire ordeal.".

Tracy Chapman, a close friend for many years, met Ms. Folbigg at the prison gates and said she had spent her first day of freedom taking pleasure in the little things.

According to Ms. Chapman, Ms. Folbigg was "in awe" of contemporary technology, such as smartphones, and these included a comfortable bed, pizza and garlic bread, and a Kahlua and coke.

She said, "Kath doesn't hate; she just wants to live the life she's missed for the past 20 years and move on.

In the Court of Criminal Appeal, Ms. Folbigg would now like to have her convictions overturned, according to her attorney Rhanee Rego.

According to Attorney General of New South Wales Michael Daley, Ms. Folbigg received the full pardon as a result of a recent investigation into her case.

All four of the children could have perished naturally, according to the investigation, which was overseen by retired judge Tom Bathurst.

A group of immunologists discovered that Ms. Folbigg's daughters all had the genetic mutation CALM2 G114R, which can result in sudden cardiac death.

There was also proof that her sons had a different genetic mutation that was connected to mice developing sudden-onset epilepsy.

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